Get Ahead of Aging, by Rebuilding Your Sleep First.

Falling asleep and staying asleep are often different problems. That’s why a strategy that helps you get drowsy at the start of your sleep window may do very little for the 2–4 a.m. stretch.Here’s one concept that can help you understand your 3 a.m. wakeups: your brain moves through 80–120 minute ultradian cycles all night long. At the end of each cycle, you get a brief arousal—often so short you don’t remember it. Those moments are normal.The question is what happens during that brief arousal: does your brain move through the transition and return to sleep, or does it tip into full alertness?Here’s how ultradian cycles, sleep stage transitions relate to3 a.m. wakeups:

A guide for midlife adults who don’t fit the “classic” apnea mold. Guest post collaboration between Chris Gouveia, MD (SleepDocs) and Kat Fu, M.S., M.S. (The Longevity Vault).Chris Gouveia, MD is a board-certified ENT surgeon and sleep apnea specialist practicing in the Bay Area of California. He completed his medical training at UCSF and his sleep fellowship at Stanford University, where co-author Kat Fu also completed her second masters degree. Dr. Gouveia treats a wide spectrum of OSA and sleep-disordered breathing, with a focus on personalized care. He shares clinical insights and evidence-based analysis on the business, tech, and finance of sleep health through his newsletter at sleepdocs.substack.com.If you picture someone with obstructive sleep apnea, the stereotype is familiar: a heavier middle-aged man, snoring loudly and nodding off at stoplights.That patient exists.But in clinic, that’s increasingly not the person I see.Instead, I see the 52-year-old marathoner who wakes up feeling like he has a hangover without drinking, the 61-year-old executive whose bloodwork looks great but who lives with constant brain fog, and the 58-year-old teacher who eats well, walks every day, does “everything right”—and still feels like sleep never truly restores her.

Let’s talk about solving it.

Raising testosterone, or estrogen isn’t enough. Sleep depends on how effectively your body can use them—and that responsiveness can be supported, even with age, here’s how:

Without addressing this layer first, every sleep strategy eventually hits a ceiling. Here’s an overlooked reason sleep routines stop working or don’t work at all (& what you can do about it):

You’ve probably seen “CBT-I” mentioned as the non-drug treatment for insomnia. Cognitive Behavioral Therapy for Insomnia (CBT-I) is a structured, skills-based approach that changes habits, thoughts, and scheduling around sleep.Insomnia becomes more common with age; roughly 20–30% of older adults live with insomnia, often for years. It’s linked with memory and concentration problems, mood changes, higher fall risk, and worse outcomes in conditions such as heart disease and chronic pain.Major guidelines, including those supported by the American Academy of Sleep Medicine, recommend CBT-I as the first-line treatment for chronic insomnia, especially in older adults where sedative-hypnotic drugs can raise fall and confusion risk.At the same time, CBT-I has trade-offs:It asks you to follow a very regular schedule and temporarily cut time in bed (“sleep restriction”), which can be tiring in the short term.It takes effort: sleep diaries, behavioral changes, and challenging long-held beliefs about sleep.Access can be limited – there are not many clinicians trained in CBT-I, and traditional one-to-one therapy is time-intensive and expensive.In this review, we’ll walk through four recent peer-reviewed studies (2023–2025) that help answer practical questions for adults in midlife and beyond:What can CBT-I actually do for sleep and daytime function?How does it seem to work in the brain?What’s realistic to expect – and by when?Who tends to benefit most, and where are the limits?How can you use this evidence to decide whether CBT-I is a good fit for you?Let’s get started.